Helicobacter pylori Containing More Phosphorylation Sites of the CagA Protein Induces Greater Reduction of Gastric Mucins

References (23)

1. 1 Lymphocytes in the human gastric mucosa during Helicobacter pylori have a T helper cell 1 phenotype [DOI]
2. 2 Helicobacter pylori CagA Induces Ras-independent Morphogenetic Response through SHP-2 Recruitment and Activation [DOI]
3. 3 Helicobacter pylori CagA Phosphorylation Status Determines the gp130-activated SHP2/ERK and JAK/STAT Signal Transduction Pathways in Gastric Epithelial Cells [DOI]
4. 4 Genome sequence analysis of Helicobacter pylori strains associated with gastric ulceration and gastric cancer [DOI]
5. 5 Higher number of Helicobacter pylori CagA EPIYA C phosphorylation sites increases the risk of gastric cancer, but not duodenal ulcer [DOI]
6. 6 Helicobacter pylori-Induced Chronic Active Gastritis, Intestinal Metaplasia, and Gastric Ulcer in Mongolian Gerbils [DOI]
7. 7 Polymorphism in the CagA EPIYA Motif Impacts Development of Gastric Cancer [DOI]
8. 8 Simple Method for Determination of the Number ofHelicobacter pyloriCagA Variable-Region EPIYA Tyrosine Phosphorylation Motifs by PCR [DOI]
9. 9 Helicobacter pylori adhesion to gastric epithelial cells is mediated by glycan receptors [DOI]
10. 10 Distribution ofvacAgenotypes inHelicobacter pyloristrains isolated from Brazilian adult patients with gastritis, duodenal ulcer or gastric carcinoma [DOI]
11. 11 Helicobacter pylori Infection Impairs the Mucin Production Rate and Turnover in the Murine Gastric Mucosa [DOI]
12. 12 Epidemiological Link between Gastric Disease and Polymorphisms in VacA and CagA [DOI]
13. 13 Helicobacter pylori cag pathogenicity island genes: clinical relevance for peptic ulcer disease development in Brazil [DOI]
14. 14 Helicobacter pylori gastritis updated Sydney classification applied in our material.
15. 15 Infection with CagA-positive Helicobacter pylori strain containing three EPIYA C phosphorylation sites is associated with more severe gastric lesions in experimentally infected Mongolian gerbils (Meriones unguiculatus) [DOI]
16. 16 Heterogeneity in the Activity of MexicanHelicobacter pyloriStrains in Gastric Epithelial Cells and Its Association with Diversity in thecagAGene [DOI]
17. 17 In Vivo Expression of Helicobacter pylori Virulence Genes in Patients with Gastritis, Ulcer, and Gastric Cancer [DOI]
18. 18 Helicobacter pylori with stronger intensity of CagA phosphorylation lead to an increased risk of gastric intestinal metaplasia and cancer [DOI]
19. 19 Secreted and Membrane-Bound Mucins and Idiopathic Peptic Ulcer Disease [DOI]
20. 20 Role of vacA and cagA in Helicobacter pylori Inhibition of Mucin Synthesis in Gastric Mucous Cells [DOI]
21. 21 Helicobacter pylori in vivo causes structural changes in the adherent gastric mucus layer but barrier thickness is not compromised [DOI]
22. 22 Effects of Cytokines, Without and with Helicobacter pylori Components, on Mucus Secretion by Cultured Gastric Epithelial Cells [DOI]
23. 23 Distribution of vacA genotypes in Helicobacter pylori strains isolated from Brazilian adult patients with gastritis, duodenal ulcer or gastric carcinoma [DOI]

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