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Noura Agarrab

Publications (4)

A Rare Presentation of Classical Homocystinuria: Cerebral Thrombophlebitis in an Adolescent

Noura Agarrab, Asmae Baaziz, Imane Zineb & Asmaa Mdaghri Alaoui · Asian Journal of Advanced Research and Reports · 2025

Homocystinuria is a rare constitutional aminoacidopathy characterized by elevated plasma and urinary homocysteine levels, most often due to cystathionine beta-synthase (CBS) deficiency, and represents the second most frequent metabolic encephalopathy after phenylketonuria. We rep...

Open access Research Article 10.9734/ajarr/2025/v19i101185

Vitamin B12 Deficiency in Infants: Clinical Manifestations, Neurological Consequences, and Therapeutic Management

Noura Agarrab, Houda El Anguoud, Asmae Baaziz, Zineb Imane & Asmaa Mdaghri Alaoui · Asian Journal of Pediatric Research · 2025

Vitamin B12 deficiency is a well-recognised cause of neurodevelopmental disorders and psychomotor regression in pediatric patients. In developed countries, infant B12 deficiency most commonly occurs in exclusively breastfed infants whose mothers have subclinical or overt B12 defi...

Open access Research Article 10.9734/ajpr/2025/v15i9473

Ataxia with Oculomotor Apraxia Type 1 Presenting in a 5-Year Old Child: Diagnostic and Clinical Considerations

Noura Agarrab, Azzeddine Laaraje, Radi Abdelilah, Soukaina Ait Hmadouch, Amal Hassani & Rachid Abilkassem · Asian Journal of Pediatric Research · 2025

Ataxia with oculomotor apraxia type 1 (AOA1) is a rare neurodegenerative disease with autosomal recessive inheritance, caused by mutations in the APTX gene encoding aprataxin, a protein involved in DNA repair. We report the case of a 5-year-old child born to consanguineous parent...

Open access Research Article 10.9734/ajpr/2025/v15i6456

Clinical Features of Classical Congenital Adrenal Hyperplasia Due to 21-hydroxylase Deficiency with Complete Virilisation

Houda El Anguoud, Noura Agarrab, Ahmed Gaouzi, Zineb Imane & Asmaa Mdaghri Alaoui · Asian Journal of Advanced Research and Reports · 2025

Background: The most prevalent form of Congenital adrenal hyperplasia (CAH) is 21-hydroxylase deficiency (21-OHD), which accounts for over 90% of CAH cases. This enzymatic defect results in decreased cortisol and aldosterone synthesis, leading to compensatory adrenocorticotropic...

Open access Research Article 10.9734/ajarr/2025/v19i81132